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Sustained activation of this pathway promotes inhibition of autophagy, metabolic reprogramming, and increased protein synthesis, thereby reinforcing senescence-associated phenotypes and contributing to the development of a proinflammatory and profibrotic microenvironment [112]

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For tobacco products, Article 5 (3) requires the submission of a list of ingredients with relevant toxicological data for the ingredients in burnt and unburnt form, as appropriate

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