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This highlights a critical gap in clinical practice, emphasizing the need for hepatologists and gastroenterologists to be more proactive in identifying smoking as a risk factor for liver disease progression and in implementing strategies to promote smoking cessation among their patients

doi:10.15585/mmwr.mm7341a2

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JNK is activated in various animal models of obesity and also in patients with NASH and its deletion results in attenuation of fatty liver (60) or nicotine plus HFD-induced hepatic steatosis in obese mice (42)