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Semaglutide presents a pronounced weight-lowering effect, mediated by direct GLP-1 stimulation of the anorexigenic (satiety inducers) proopiomelanocortin (POMC) and cocaine- and amphetamine-regulated transcript (CART) neurons along with indirect inhibition of orexigenic neurons (appetite inducers) neuropeptide Y (NPY) and agouti-related peptide (AgRP), located in the arcuate nucleus (ARC) in the hypothalamus, which is vital in the regulation of appetite and energy balance [114,115,116]

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However, there is currently no FDA-approved indication for GLP-1 receptor agonists specifically for inflammation, and the optimal dosing strategy for anti-inflammatory effects has not been established through rigorous clinical trials

The potential mechanism for this effect includes the deposition of collagen through the ERK1/2 signaling pathway, re-epithelialization and glandular tissue formation [3]